Are Plastic Nursing Bottles Safe for Babies? What Studies Show

According to the results of five studies, compared with non-PC baby bottles, there is more BPA residual leached from PC (polycarbonate) bottles with the temperature and incubation time. It is suggested that glass baby bottles are a better choice than PC-bottles.  However, if you do use PC-baby bottles, even though BPA residual is leached from the bottle at higher temperatures during the process of sterilization and boiling, it doesn’t exceed the tolerable level. As for excessive exposure to BPA and its influence, it has been shown that prenatal BPA exposure is related to children's external behavior problems (including hyperactivity and aggression) at age 2. Continual exposure to BPA in both the prenatal and postnatal stages can lead to obesity and hyperlipidemia in the child.

Created on November 21 2015 at 08: 00 AM


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Details of Scientific Answers: Click on each bullet to Read References

References:

1: "Prenatal Bisphenol A Exposure And Early Childhood Behavior," Environmental Health, 2009, by JM Braun, K Yolton, KN Dietrich, R Hornung. (Citations: 258).

Background: Prenatal exposure to bisphenol A (BPA) increases offspring aggression and diminishes differences in sexually dimorphic behaviors in rodents. Objective: We examined the association between prenatal BPA exposure and behavior in 2-year-old children. Methods: We used data from 249 mothers and their children in Cincinnati, Ohio (USA). Maternal urine was collected around 16 and 26 weeks of gestation and at birth. BPA concentrations were quantified using high-performance liquid chromatography–isotope-dilution tandem mass spectrometry. Child behavior was assessed at 2 years of age using the second edition of the Behavioral Assessment System for Children (BASC-2). The association between prenatal BPA concentrations and BASC-2 scores was analyzed using linear regression. Results: Median BPA concentrations were 1.8 (16 weeks), 1.7 (26 weeks), and 1.3 (birth) ng/mL. Mean (± SD) BASC-2 externalizing and internalizing scores were 47.6 ± 7.8 and 44.8 ± 7.0, respectively. After adjustment for confounders, $log_{10}$ -transformed mean prenatal BPA concentrations were associated with externalizing scores, but only among females [β = 6.0; 95% confidence interval (CI), 0.1-12.0]. Compared with 26-week and birth concentrations, BPA concentrations collected around 16 weeks were more strongly associated with externalizing scores among all children (β = 2.9; 95% CI, 0.2-5.7), and this association was stronger in females than in males. Among all children, measurements collected at ≤ 16 weeks showed a stronger association (β = 5.1; 95% CI, 1.5-8.6) with externalizing scores than did measurements taken at 17-21 weeks ( β = 0.6; 95% CI, -2.9 to 4.1). Conclusions: These results suggest that prenatal BPA exposure may be associated with externalizing behaviors in 2-year-old children, especially among female children.


References:

1: "Perinatal And Postnatal Exposure To Bisphenol A Increases Adipose Tissue Mass And Serum Cholesterol Level In Mice," Journal of atherosclerosis and thrombosis, 2007, by J Miyawaki, K Sakayama, H Kato. (Citations: 160).

Aim: To investigate whether the perinatal and postnatal exposure of mice to bisphenol A (BPA) caused the development of obesity and/or hyperlipidemia. Methods: Pregnant mice were exposed to BPA in drinking water at concentrations of either 1 µg/mL (LD group) or 10 µg/mL (HD group) from gestation day 10 and throughout the lactating period. After weaning, the pups were allowed free access to drinking water containing the appropriate concentrations of BPA. The body weight, adipose tissue weight, and serum lipid levels were measured in the offspring at postnatal day 31. Results: In females, the mean body weight increased by 13% in the LD group (p<0.05) and 11% in the HD group (p<0.05) compared with the control group. The mean adipose tissue weight increased by 132% in the LD group (p<0.01). The mean total cholesterol level increased by 33% in the LD group (p<0.01) and 17% in the HD group (p<0.05). In males, the mean body weight and mean adipose tissue weight increased by 22% (p<0.01) and 59% (p<0.01), respectively, in the HD group compared with the control group. The mean triacylglycerol level increased by 34% in the LD group (p<0.05). Conclusions: The continuous exposure of mice to BPA during the perinatal and postnatal periods caused the development of obesity and hyperlipidemia.


References:

1: "Migration Of Bisphenol A From Plastic Baby Bottles, Baby Bottle Liners And Reusable Polycarbonate Drinking Bottles," Food Additives & Contaminants, 2009, by C Kubwabo, I Kosarac, B Stewart. (Citations: 76).

Human exposure to bisphenol A (BPA) has recently received special attention. It has been shown that exposure to BPA may occur through the consumption of beverages or foods that have been in contact with polycarbonate (PC) plastic containers or epoxy resins in food ...


References:

1: "Increased Migration Levels Of Bisphenol A From Polycarbonate Baby Bottles After Dishwashing, Boiling And Brushing," Food Additives & Contaminants, 2003, by C Brede, P Fjeldal, I Skjevrak. (Citations: 233).

Baby bottles are often made of polycarbonate plastic. Impurities remaining in the bottle from the monomer bisphenol A can migrate from the plastic bottles into baby food, thereby causing a health concern. Previous migration testing of new baby bottles showed only ...


References:

1: "Migration Of Bisphenol A From Polycarbonate Baby Bottles Under Real Use Conditions," Food Additives & Contaminants, 2008, by NC Maragou, A Makri, EN Lampi. (Citations: 74).

Migration of the potential endocrine disrupter, bisphenol A (BPA), from 31 polycarbonate (PC) baby bottles into aqueous food simulants was studied under real repetitive use, using a sensitive and fully validated liquid chromatographic method with fluorescence detection. ...



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